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Summary
➡ The second most common cause of dementia, known as vascular dementia, is due to microscopic bleeding in the brain. Alzheimer’s is the most common cause, and the two main drugs used for treatment are Kisunla and Leqembi. However, these drugs have been found to cause microscopic bleeding in the brain, which is a major cause of vascular dementia. Therefore, the treatment for Alzheimer’s could potentially worsen dementia symptoms by causing additional brain bleeding.
➡ The article discusses the potential dangers of Alzheimer’s and dementia drugs, specifically Kasunla and Leqembi. These drugs, while intended to slow the progression of dementia, have serious side effects including brain bleeding, which can worsen dementia symptoms and even be fatal. The author suggests asking your doctor about these risks before starting treatment. The article ends by hinting at safer, natural alternatives to these drugs, such as vitamin D supplementation.
➡ Vitamin D and Vitamin K2 may play a significant role in preventing and treating Alzheimer’s and dementia. Studies have shown that Vitamin D can help clear harmful amyloid plaques in the brain, and a deficiency in Vitamin D has been linked to a higher risk of these diseases. Similarly, Vitamin K2 has been found to reduce brain inflammation, improve vascular health, and decrease amyloid plaques. Therefore, maintaining sufficient levels of these vitamins could potentially lower the risk of developing Alzheimer’s and dementia.
➡ Warfarin, a blood thinner, can block vitamin K2 and cause major or fatal bleeding, potentially worsening dementia. Vitamin K2 and magnesium have been found to potentially reduce brain inflammation and degeneration, and may help prevent Alzheimer’s disease. Depression is common in Alzheimer’s patients and vitamin K2 may help prevent it. Maintaining adequate magnesium levels can support cognitive health and potentially slow the progression of Alzheimer’s disease.
➡ Studies show that maintaining a healthy intake of magnesium and taurine can help prevent dementia, especially in older adults and those with metabolic risk factors. Magnesium is crucial in slowing down neurodegeneration processes, while taurine has shown potential in improving cognitive deficits caused by Alzheimer’s disease. Both supplements have been found to protect against brain damage and improve memory and cognitive functions. It’s important to consult with your doctor before starting any new supplement regimen.
Transcript
Have your cameras ready. You can scan the QR code. Make sure you put in your email address so we can notify you if you you would like to stay up to speed on our once a week health and wellness podcast, helping to make you aware of the hidden dangers of pharmaceuticals and Big Pharma and restoring at the same time your faith in nature, which is all this platform’s about. All right, so Here comes the PowerPoint. Let me bring it up on the screen. I hope you enjoy. Anybody out there who is worried about keeping their cognition, their brain working and functioning as optimally as possible for as long as possible, this show is for you.
This is not just for Alzheimer’s and dementia patients. However, there’s millions of them that have been diagnosed with dementia, memory deficit and Alzheimer’s. And this is for all of you and all of them, they have got to know this information. You need to know this information before your MD decides to prescribe to you the drugs they prescribe for dementia and Alzheimer’s. You need to know what they know about those drugs if you want to keep your mind. All right, here we go. We’re going to dive in how to not lose your mind, especially in response to Alzheimer’s and dementia.
Here’s the QR code. Scan it. You scan it. Put in your email. That’ll take you to our website. The PowerPoint to this presentation can be found and downloaded for free. Every slide you’re about to see can be downloaded in the resource tab at the Dr. Ardis Show. QR code will take you there. It’ll also allow you to put in your email so you can be notified with my next weekly podcast and the health topic we are going to be educating you on and giving you hope in. All right. According to the Alzheimer’s Association, Alzheimer’s Worldwide, 55 million people are living with Alzheimer’s and other dementia concerns.
Quick facts, here’s just some over 7 million Americans are living with Alzheimer’s. By the year 2050, the number is expected to rise to nearly 13 million. So if you don’t watch this podcast and you ain’t sharing this with other people and you don’t want to be a victim of Alzheimer’s and losing your mind, you better pay attention to this. Because if you don’t, I’m telling you, you, you are going to increase the risk of developing dementia by the year 2050 or being diagnosed with Alzheimer’s. One in nine people age 65 and older has Alzheimer’s health. And long term care costs for people, $3.84 billion.
Actually, that’s not 0.8. That’s $384 billion in 2025. And they expect by the year 2050, it’s going to cost $1 trillion a year to take care of those with Alzheimer’s and dementia. The lifetime risk for Alzheimer’s at age 45 is 1 in 5 for women and 1 in 10 in men. Sorry, ladies. Nearly 12 million Americans provide unpaid care for people with Alzheimer’s or other dementias. In 2024, unpaid caregivers provided an estimated more than 119 billion hours of care valued at more than $413 billion. So does this affect you? Yeah. Does it affect our economy? Yes.
Does it affect our lives? You better believe it. Nearly four in five Americans would want to know if they had Alzheimer’s disease before having symptoms. Supposedly. According to alz.org which is short for Alzheimer’s, 92% of Americans would want to take a medication that could slow the progression of Alzheimer’s per surveys. Give me the drugs. I’m about to show you those drugs and oh, my God, you don’t want those drugs. You just think you do. Nearly three in five Americans said they would accept moderate or very high levels of risk with taking medications to slow the progression of Alzheimer’s disease.
I’m sorry, you three and five Americans who are like, I don’t care what the risks are. What if I told you it is published that all the drugs they prescribe for Alzheimer’s and dementia are known to cause worsening Alzheimer’s and dementia. And do you know how many percentages of people that affects. I’m about to show you if you like those risks. So absurd. And then at the end of this presentation, I’m going to show you all the natural antidotes that are proven to prevent Alzheimer’s and dementia that your FDA approval process and your medical doctors are not going to tell you.
What is Alzheimer’s disease. According to the Alzheimer’s Association, Alzheimer’s disease is a type of dementia that affects memory, thinking and behavior. Symptoms eventually grow severe enough to interfere with daily tasks. Alzheimer’s is the most common cause of dementia, a general term for memory loss and other cognitive abilities serious enough to interfere with daily life. Alzheimer’s disease accounts for 60 to 80% of dementia cases. What? What accounts for the others? I’ll show you. Alzheimer’s is not a normal part of aging. According to alz.org the greatest known risk factor is increasing age. Oh, wait, wait. What did the statement before that say? Alzheimer’s is not a normal part of aging.
Yet the greatest known risk factor is increasing age. Okay, that’s literally reads like an oxymoron. And the majority of people with Alzheimer’s are 65 and older. Alzheimer’s worsens over time. Alzheimer’s is a progressive disease where dementia symptoms gradually worsen over a number of years. You’re about to find out why. In its early stages, memory loss is mild. But with late stage Alzheimer’s, individuals lose the ability to carry on a conversation and respond to their environment. On average, a person with Alzheimer’s lives four to eight years after diagnosis, but can live as long as 20 years, depending on other factors.
Alzheimer’s has no cure, but there are two treatments. Donanemab, which is a monoclonal antibody with a brand name Casunla, and losanimab, otherwise known as Leqembi, demonstrate that removing beta amyloid, one of the hallmarks of Alzheimer’s disease. Y’ all have heard of amyloid plaques? They call them beta amyloid, one of the hallmarks of Alzheimer’s disease, from the brain reduces cognitive and functional decline in people living with early, early Alzheimer’s. Remember these two drugs? Look at the pictures. These are the two drugs, the most common you give for Alzheimer’s, which is the number one cause for all dementia in the world.
80% of you Alzheimer’s in the brain. The brain has 100 billion nerve cells called neurons. Each nerve cell connects with many others to form communication networks. Groups of nerve cells have special jobs. Some are involved in thinking. Some are involved in learning and remembering. Others help us see, hear and smell. Scientists believe Alzheimer’s disease prevents part of a cell’s factory from running. Well, they are not sure where the trouble starts. I’m going to show you where it starts. But just like a real factory, backups and breakdowns in one system cause problems in other areas. As damage spreads, cells lose their ability to do their jobs and eventually die.
Causing irreversible changes in the brain. Irreversible changes according to Alzheimer’s association. What is dementia? Dementia is a general term for loss of memory, language, problem solving, and other thinking abilities that are severe enough to interfere with daily life. Alzheimer’s is the most common cause of dementia. About dementia, I know these seem like they’re interchangeable, Alzheimer’s and dementia, but they are distinct. It just so happens Alzheimer’s is the number one diagnosed cause for dementia. Dementia is not a single disease. It’s an overall term to describe a collection of symptoms that one may experience if they are living with a variety of diseases, including Alzheimer’s.
Dementia is an umbrella term for loss of memory and other thinking abilities severe enough to interfere with your daily life. And to the right, you can see what types Alzheimer’s, vascular, Lewy body frontotemporal, and other, including Huntington’s disease. Or there could be mixed dementia, meaning dementias from more than one cause. Alzheimer’s disease accounts for 60 to 80% of the cases. Pay attention to this next sentence. Vascular dementia, which occurs because of microscopic, really tiny bleeding, blood vessel blockage in the brain, is the second most common cause of dementia. Go to the bottom. Dementia is often incorrect.
Oh, sorry. Let’s go to the next sentence. There are many other conditions that can cause symptoms of cognitive impairment but that aren’t dementia, including some that are reversible, such as thyroid problems and vitamin deficiencies. Dementia is often incorrectly referred to as senility or senile dementia, which reflects the formerly widespread but incorrect belief that serious mental decline is a normal part of aging. All right, we’re going to go back to the top of the screen. Sorry. You all better listen to me. We’re going to pay attention to the first two sentences. Alzheimer’s disease accounts for 60 to 80% of cases.
Vascular dementia, which occurs because of microscopic bleeding, little bitty bleeding in the brain, and blood vessel blockage like plaque in the brain, is the second most common cause of dementia. I’m going to focus on a term here, microscopic bleeding in the brain. Now stay there with me. I’m going to show you why it’s so important. These first two sentences literally set the stage for the rest of this presentation, which I still have, like, 70 more slides to show you, so pay attention to this one slide. This is so important. I’m gonna break this down even further.
Vascular dementia, which occurs because of microscopic bleeding in the brain, is the second most common cause of dementia. What’s number one? Alzheimer’s. What do they get for Alzheimer’s? And dementia, two primary drugs. I showed them earlier. Then they’re telling you here, outside of Alzheimer’s, which is up to 80% of all reasons for dementia, there’s this second cause for dementia known as vascular dementia, caused by microscopic bleeding in the brain. So I just want to leave this sentence on the screen. Vascular dementia, the second most common cause for losing your mind, is also caused by microscopic bleeding in the brain.
Now, this is the Cleveland Clinic. You see the word hemorrhage there. I’m not sure all of y’ all know what that word means, so I have to show you what the Cleveland Clinic means by the word hemorrhage. This is important for the rest of the presentation. A hemorrhage is bleeding from a damaged blood vessel. Many things can cause bleeding inside and outside of the body. Stay with me. Hemorrhage. A hemorrhage is bleeding from a damaged blood vessel. If you look at the word above for vascular dementia, that’s described as microscopic bleeding in the brain. Another word for microscopic bleeding in the brain would be micro hemorrhaging.
In the brain, hemorrhaging means bleeding. Microscopic bleeding means microscopic hemorrhaging. They are synonymous. They are the same terminology. This is very important. Do not forget this statement. Vascular dementia, which is the second most common cause of dementia, occurs because of microscopic bleeding in the brain, otherwise known as microscopic hemorrhaging. Don’t forget that statement. I’m just going to leave it there, but I’m going to come back to it. Here are the signs and symptoms of dementia going back to alz.org symptoms and signs of dementia include short term memory, keeping track of a purse or wallet, paying bills, planning and preparing meals, remembering appointments, traveling out of the neighborhood.
Dementia symptoms are progressive, which means that the signs of cognitive impairment start out slowly and gradually get worse over time, leading to dementia. Dementia treatment and care Treatment of dementia depends on its cause. In the case of most progressive dementias, including Alzheimer’s disease, there is no cure. But there are two treatment options, Kasunla and Leqembi, which are these two drugs on the right. You saw these for Alzheimer’s. They’re also prescribed for dementia. Now let’s look at these two drugs that they are prescribing for dementia and Alzheimer’s and see what it actually causes. This should shock you.
I didn’t know this until we built out this presentation. All right, so the first drug for Alzheimer’s and dementia I just showed on the screen is brand name Kisunla K I S U N L A. So if you know somebody with Alzheimer’s or you know somebody with dementia, they’re on this drug, most likely, or the next one. So Kisunla, otherwise known as generic name Donanemab, mab, means monoclonal antibodies on the end. Donain, mab, Donanamab, otherwise known as Kusinlu. This drug you see at the bottom is used for Alzheimer’s to help slow down the decline in memory, thinking and daily function according to drugs.com.
now to the right, you’re going to see that this drug has a FDA black box warning. And let’s read it together. Amyloid related imaging abnormalities. Just so y’ all know, they only diagnose Alzheimer’s from dementia, including amyloid plaques in the brain. They’re telling you this drug causes amyloid related imaging abnormalities. More amyloid plaques are going to show up in your future brain scans because of the drug. That’s not the craziest thing. I have to show you a second statement. Amyloid related imaging abnormalities, abbreviated aria, typically presents without symptoms but can cause rare life threatening events such as intracerebral hemorrhages.
The Alzheimer’s drug, called Kunla or Donanemab, causes intracerebral in the brain bleeding. And that’s an FDA black box warning, the highest warning they can give you for any drug. All right, so let’s just stay there. I’m just going to highlight that statement. Do you remember? Let’s read it together. A, R, I, A. Amyloid imaging abnormalities. Typically presents without symptoms but can cause rare life threatening events such as intracerebral hemorrhaging. Remember what the word hemorrhaging means? It means bleeding. Remember the statement from earlier? Vascular dementia, which occurs because of microscopic bleeding in the brain, is the second most common cause of dementia.
Holy crap. Did you know? The number one prescribed drug for Alzheimer’s which includes dementia as a symptom, is published with a black box warning that it increases the risk of micro hemorrhaging in the brain that is the second most common cause, dementia. If you want worsening dementia, you might want to ask for this drug. Common Donanemab side effects. Common Casunla side effects include headache and swelling in areas of the brain with or without small spots of bleeding in or on the brain surface. These occurred in at least 10% of patients and at a higher incidence compared to the placebo.
Oh my God. They know. One in 10. Actually, more in at least 10%. That means there’s more than 10% of people who get this drug for Alzheimer’s or dementia will have brain bleeds. Which is the second most common cause for dementia. Oh my God. Remember this statement, I’m going to show this a million times till it gets drilled into your brain. They tell you Alzheimer’s is the number one cause for dementia representing 60 to 80%. The next statement is vascular dementia like blood flow dementia, which occurs because of microscopic bleeding and blood vessel blockage in the brain is the second most common cause of dementia.
The drug for Alzheimer’s causes microscopic bleeding in the brain in more than 10% of people that get it. More serious side effects. Remember the effect I just showed you is common side effects. And it is very common for people to have brain bleeds on our drug for Alzheimer’s which is a cause for the second highest amount of dementia. That’s not even the serious side effects. Okay, let’s look at the serious side effects they list on drugs.com for this Alzheimer’s dementia drug. Serious allergic reactions include swelling of the face, lips, mouth or eyelids, difficulty breathing and hives.
Infusion related infect reactions injecting inside of you. If you you have an infusion related reaction, your infusion may be slowed down or stopped. Tell your healthcare provider right away if you have symptoms of these with that drug. Sweating, irritation of the skin, headache, nausea, chest pain, vomiting or problems breathing during an infusion. Ask your doctor if this drug’s right for you. All right, so up to 10% of people also on this drug will experience anaphylactic reactions. This is also from drugs.com the same drug for Alzheimer’s and dementia. Immunologic. They risk they list. It is very common.
The very common category. People on this drug will develop anti drug antibodies. That means as you take drugs your body’s gonna make antibodies against them. We call this autoimmune disease and inflammatory reactions according to the nervous system at the bottom. These are continued known very common side effects of these drugs for Alzheimer’s and dementia. Kassunla and the monoclonal antibody. Very common. Look at this. A RIA H micro hemorrhage. In 25% of everybody that took this drug in clinical trials experienced hemorrhaging in the brain which is the second most common cause for dementia in the whole world.
Superficial siderosis. And I will show you what that means in just a minute. And then headaches. And 13% patients have had both micro hemorrhage and superficial siderosis. Siderosis. And I’ll show you what that means in a second. Don’t worry, it’s on the next slide. We’re going to take this nervous system we’re going to take this nervous system and we’re going to move it to the top of the next slide. That whole thing, move it to the top. Very common. Will experience microhemorrhage. All right, ready? Watch this. Do you remember the statement this is common donanemab side effects.
Remember this statement from earlier? Vascular dementia, which occurs because of microscopic bleeding in the brain is the second most common cause of dementia. The drug for Alzheimer’s is published that in 25% of everyone in the clinical trials taking this drug experienced micro hemorrhaging bleeding in their brain. One in four people on this drug will develop internal brain bleeds. That is the second most common cause. So if you got Alzheimer’s, let’s add brain bleeding to you by prescribing Donanemab Kasunla to you as an Alzheimer’s patient. Disgusting. Ask your doctor if dementia is right for you. Worsening dementia and microbleeds in your brain.
Now, patients on this drug have had both micro hemorrhaging in the blaine that leads to worsening dementia and superficial siderosis. What is that? I’m going to show you superficial siderosis, a rare neurodegenerative disease caused by an iron deposition on the brain. Okay. Actually, it’s caused by the drugs for Alzheimer’s and dementia I just showed you. That is a published side effect of the drug. The iron is toxic to the nerves and neural tissues and will cause damage over time. A chronic bleed into the cerebral spinal fluid, often from previous surgery or trauma, is what leads to iron deposition.
No, it doesn’t. The drugs for Alzheimer’s and dementia I just showed you on drugs.com is a published side effect on the package insert found in the physician’s desk reference for the drug prescribed for Alzheimer’s and dementia. It causes this. That means the drug is causing damage to your blood brain barrier and allowing iron to get into your brain to cause worsening neurological and neurodegenerative effects. Ask your doctor if metals in the brain are better for you when they prescribe those drugs. Here’s highlighting information from the package insert for Kasunla. Kasunla’s injection drug. Actually, let’s just go to the black box warning.
We’re going to blow it up and highlight a few statements warning, amyloid related imaging abnormalities, which we already highlighted. Serious and life threatening events can occur. Aria. Amyloid related imaging abnormalities can be fatal. FDA wants to make sure. You know, you could die from this drug. It can kill you. Serious intracerebral hemorrhages, serious bleeding in your brain have occurred in patients treated with this class of medications. A riae can cause focal neurological deficits that can mimic ischemic strokes. Holy crap. The drugs for Alzheimer’s and dementia can make you have what appears to be a stroke. Like your symptoms.
You can’t talk, slurring, your speech, can’t move one side of your body. That can just happen to you. It mimics a stroke, not that you had a stroke. The drug itself can make you lose control of your body, parts of your body that make it look like a stroke. Ask your doctor if a false stroke, a pseudo stroke or brain bleeds are right for you and if that will help you with your future dementia prevention efforts, which is why you went to your medical doctor in the first place. Let’s look further into this package. Insert for Kasunla.
The most common serious adverse reactions, at least 10% and higher, experienced micro hemorrhaging, superficial siderosis. I just showed you iron deposition in the brain and headaches. I just moved to the top and put the drug up there so you can see it. Most common adverse reactions, more than 10% have had micro hemorrhaging in their brain. Remember the statement from earlier. Vascular dementia, which occurs because of microscopic bleeding in the brain, is the second most common cause of dementia. This is the next drug they want you to know are the most prescribed drugs. This is the second of the two.
Leqembi L, E, Q, E, M, B, I is the brand name. Losanimab, another monoclonal antibody, is the generic name of the drug. So there’s the brand name and that’s what it looks like. Oh, let’s go back. Sorry. Let’s read the statement at the bottom. This is used for Alzheimer’s disease to help slow the progression of dementia in patients who have mild cognitive impairment, MCI or mild dementia symptoms. Licinimab infusion works by reducing amyloid beta plaques and protofibrils in the brain, which helps slow down the worsening of dementia symptoms. Oh, thanks. Maybe this one’s better than the other one.
Hold up. Let’s read together here. Let’s. This is a black box warning. Let’s read together slowly, shall we? This is the other second most prescribed drug for Alzheimer’s and dementia. Serious intracerebral hemorrhages, some of which have been fatal, have been observed in patients treated with this class of medications. Let’s keep it on the screen. Do you remember this statement from earlier? Vascular dementia, which occurs because of microscopic bleeding in the brain, is the second most common cause of dementia. Holy crap. The second most prescribed drug for Alzheimer’s and dementia patients, called Lisanumab or Leqembi, causes the second most common cause of dementia, vascular dementia.
It causes serious intracerebral bleeding. Read the statement with the arrow. Intra brain bleeding, some of which have been fatal, have been observed in patients treated with this drug. So when they say your loved one died from Alzheimer’s, holy crap. How do you know they died from Alzheimer’s? The drugs they had your Alzheimer’s loved one on can be fatal. The drug itself can kill you, yet they blame it on the disease. I’m sorry, that’s a black box warning by the fda. The most serious warning they can give you for a drug that the drug’s fatal. Did your medical doctor tell you that when they gave it to you? Did they tell you that the drugs.
Each of the drugs, generic and brand name, did they tell you that each drug for Alzheimer’s and dementia are published to cause worsening bleeding in the brain? They both have black box warnings telling you that’s going to happen. And did they tell you that you could die from it? Here’s a list on drugs.com for Leqimbi. What are its most common side effects? Up to 10% of people on that drug in the trials experienced atrial fibrillation from the drug. So heart disease, 10%. Diarrhea, liver lymphopenia, lower white blood cells, so a reduced immune system, 20%. Infusion related reactions, which I’m not going to define those.
Don’t inject it, obviously respiratory. Up to 10% of you’re going to develop a chronic cough and then the nervous system. Amyloid related imaging abnormalities. 10% of you are going to experience it. 1 in 10. We’re going to keep going on drugs dot com. They state this medicine may cause amyloid related imaging abnormalities. Aria. Which can be seen as temporary swelling or bleeding in the brain. Oh, my God. There it is again. Bleeding of the brain that causes dementia. Call your doctor right away. If you have changes in vision, confusion, dizziness, headache, nausea or seizures. Then they go on to say this medicine may cause infusion related reactions which can be life threatening.
Thank you. Go ahead and inject that into my body. I know it might kill me. Thanks. This medicine may cause serious allergic reactions, including anaphylaxis and angioedema. Swelling of your eyelids, nose, lips, ears, vagina, you name it. Penis. Which can be life threatening and require immediate medical attention. This is what you need to know about the drugs. They’re prescribing all of you for dementia and Alzheimer’s. Now, let’s stay focused on the top one. I cannot stop doing this. It’s so ridiculous. Remember the statement from earlier? Vascular dementia, which occurs because of microscopic bleeding in the brain, is the second most common cause of dementia.
The drug leqembi is known to cause temporary swelling or bleeding in the brain. Ask your doctor if bleeding in the brain is right for you. Now let’s go to the package insert for Leqembi. This is the intravenous one. All right. Leqembi can cause amyloid, which are the plaques for Alzheimer’s. The drugs themselves, both of them can cause amyloid related imaging abnormalities. You’re going to see changes in the amount of amyloids in your brain. Understands? Yeah. Because the drugs cause more of them then they tell you. Serious and life threatening events can occur. Serious intracerebral hemorrhaging, not slight, not minuscule.
No. Serious intracerebral inner brain hemorrhaging or bleeding greater than 1cm have occurred in patients treated with this class of medications. Oh, thank you. Let’s cause some more bleeding. The most common adverse reactions of leqemb at approximately 10% and higher. What if it’s 90%? What if it’s 50%? I don’t know. They just tell you it’s higher than 10%. Infusion related reactions, Amyloid related imaging abnormality, microhemorrhages. Amyloid related imaging abnormality, edema, effusion and headaches. I’m highlighting the word micro hemorrhages. Remember Cleveland Clinic, hemorrhage means bleeding, internal bleeding. When your doctor prescribes any of these drugs for Alzheimer’s or dementia, Kisunla or Leqembi, I would like you to ask your doctor if brain bleeds that these drugs cause, which are the second highest cause of dementia, are right for your brain.
Now, how to treat and deal with Alzheimer’s and dementia. Naturally, I think there’s probably a billion other ways you could treat Alzheimer’s. Mild cognitive decline, dementia. Then giving you a drug, FDA approved, approved, with a black box warning for each that says your dementia can get worse or you could die from the hemorrhaging effects in the brain of these drugs. Oh, which by the way, also caused the second cause, second most common cause for dementia. This is why the doctor Artist show exists. That’s why I ever started speaking out in the first place. All right, so per the Alzheimer’s association, this was published by them.
So ridiculous. All right, this is a NIH study. It’s on pubmed.gov vitamin D supplementation an incident of dementia effects of sex, APOE and baseline cognitive status Currently, more than 50 million people around the world live with dementia. This we’re now getting into the solutions and hope of other options versus those that could kill you and cause you to have brain bleeds that worsen your dementia. These are our published medical science papers that show you nature is much safer than the prescriptions. They expect this number to triple to 50 million with dementia and Alzheimer’s by the year 2050.
They’re just referencing that again. Vitamin D deficiency may be a modifiable risk factor and has been recognized as a widespread health problem with a worldwide prevalence for up to 1 billion people. Vitamin D is known to participate in the clearance of amyloid beta aggregates. Holy crap. Did you know vitamin D can destroy and get rid of amyloid plaques in Alzheimer’s patients brains? Did your medical doctor tell you that when they prescribe those drugs that can make you have more amyloid plaques in your brain? I just showed you all the black box warnings. If they’re not, if your neurologist is not prescribing vitamin D, fire them.
Vitamin D is known to participate in the clearance of amyloid beta plaque, one of the hallmarks of Alzheimer’s disease and may provide neuroprotection against alpha sorry alpha beta induced taurine hyper phosphorylation. Low levels of serum vitamin D have been associated. Low levels of blood of vitamin D have been associated with a greater risk of dementia and Alzheimer’s disease. Did you know that? Did your neurologist tell you that? Okay. A recent randomized clinical trial in 200 in 210 patients with Alzheimer’s disease assess the effect of 12 month vitamin D supplementation on cognition and betaplaques, reporting improved performance on several cognitive tests and lower amyloid beta plaques burdens.
Wow. Science has already proven this. Several observational studies have provided evidence on the association of lower vitamin D with greater risk of Alzheimer’s disease and dementia and the positive effect of vitamin D supplementation on performance on neuropsychological tests and the risk of developing Alzheimer’s disease. While exposure to vitamin D was associated with significantly lower dementia incidence in both males and females, the sex specific difference was also statistically significant. Pay attention here. Females, the effect of vitamin D exposure was greater in females than in males. The lower the vitamin D level in females, the higher amounts of dementia.
But in both sexes it was both significant. It was just worse for females. The effect of vitamin D exposure was greater there in females and in males associations of estrogen in activated vitamin D and declining levels of estrogen in aging females. Evidence has shown that estrogen may increase the activity of enzymes responsible for activating vitamin D. Subsequently, it can be hypothesized then that declining levels of estrogen in peri and postmenopausal stages could contribute to vitamin D deficiency in females as the estrogen levels are dropping because of perimenopause and menopause postmenopause. This could be why you all need more vitamin D and why females are more impacted by dementia with lower levels of vitamin D.
Female participants were postmenopausal and we speculate, more likely to have low levels of the activated form of vitamin D due to lower estrogen levels in our study, while exposure to vitamin D was associated with significantly lower incidence of dementia in both apoe. Sorry, I didn’t even define that acronym. APOE carriers and non carriers the effect was greater in non carriers must be a gene. Similar findings have been observed in the past explained by less vitamin D deficiency and APOE carriers which must be a specific gene for dementia and Alzheimer’s. This gene carriers may have higher concentrations of circulating vitamin D compared to non carriers despite the same intake due to greater intestinal absorption of dietary vitamin D and lower kidney excretion.
So they’re telling you vitamin D is correlating all these issues even with genetics Neurology the medical journal titled Neurology which Alzheimer’s dementia has to do with nerve nervous system vitamin D and the risk of dementia and Alzheimer’s disease 1658 elderly ambulatory adults those that can walk free from dementia, cardiovascular disease and stroke who participated in the US Population Based Cardiovascular Health Study between 1992 and 1993 and 1999 were included in this study during an average follow up of 5.6 years. So they’ve the average time frame of following all these elderly people that could walk without any of these diseases.
171 participants developed all cause dementia including 102 cases of Alzheimer’s disease using Cox proportional hazards models. The multivariate adjusted hazard ratios for incident all cause dementia in participants who were severely vitamin D deficient the multivariate adjusted hazard ratios for incidence of Alzheimer’s disease in participants who were severely vitamin D deficient and deficient compared to participants with sufficient concentrations. This is what they were evaluating in multivariate adjusted penalized smoothing ALPINE plots the risk. You can ignore all that scientific jargon of how they write these data points, so let’s just start. The risk of all cause dementia and Alzheimer’s disease markedly increased below a threshold of 50 nanomoles per liter of vitamin D in your blood.
The lower your vitamin D level below that 50 mark, the higher your risk factor for developing Alzheimer’s and dementia. Their conclusions are this. Our results confirm that vitamin D deficiency is associated with a substantially increased risk of all cause dementia and Alzheimer’s disease. Oh, guess what else? Vitamin D supplementation has not ever done caused intra brain bleeding or been fatal. Oh, but the drugs for Alzheimer’s and dementia that are FDA approved and you can find and get from a prescription pad of your from your medical doctor and go to CVS and Walgreens and start ingesting every day can kill you and can make more amyloid plaque show up in your brain.
A ria remember that term. And can cause micro bleeding in your brain which is the second cause for almost all Alzheimer’s and dementia patients. All right, so now it’s vitamin D. And now let’s go to vitamin K2. This is the Nutrients Medical Journal. This is published in 2021. Vitamin K2 holds promise for Alzheimer’s prevention and treatment. Let’s see what they found in this study. Oh, here’s vitamin K2 in the middle. I like infographics. They’re simple to follow. Let’s go bottom left. Vitamin K2 rescues mitochondrial function. Oh, that’s all. That’s just the batteries for every cell in your brain and everywhere else in your body.
Vitamin K2 decreases brain inflammation called neuroinflammation. Congratulations. Vitamin K2 is has antioxidant effect. That’s great. Antioxidants lead to inflammation in all diseases. Vitamin K2 decreases amyloid plaques. Vitamin K2 improves vascular health. Vitamin K2 could connect microbiome and Alzheimer’s. Vitamin K2 impacts Parkinson’s and multiple sclerosis. However, unlike vitamin K1, vitamin K2 activates a variety of outside the liver vitamin K properties which have many complex roles. Of these vitamin K D vitamin D, the best studied are the matrix GL A1 protein osteocalcin which which helps with bone density and growth arrest. Specific protein MGP inhibits the calcification of soft tissue and is present in the arterial wall.
Osteocalcin is important for bone health and recent research has highlighted its additional roles in brain signaling, energy metabolism and inflammation. Gas 6 is widely expressed in the nervous system and it regulates neuroinflammation and cancer cell signaling that might be important. You need to know this because that’s what vitamin K activates. The anti apoptic and antioxidant effects of vitamin K2 we will first consider the effects of vitamin K2 and Gas 6 on beta amyloid plaques. Beta amyloid plaques leads to neuronal death both by promoting apoptosis cell death of brain cells and by direct toxicity. In PC12 cells derived from rat pheochromocytoma.
Vitamin K2 prevented neuronal death resulting from amyloid plaques, the most neurotoxic form of amyloid beta plaque. When cells were exposed to either hydrogen peroxide or out or amyloid plaques, the cells that were pre treated with vitamin K2 exhibited markedly less cell death as measured by flow cytometry. Sorry, sometimes I’m reading too fast. Pretreatment with vitamin K2 also decreased the amount of cell death signaling proteins. It reduced the presence of reactive oxidative species reducing oxidative stress and it increased the amount of the greatest antioxidant the human body can make in the liver called glutathione. The authors identified the inactivation of P38 map kinase pathway as a mechanism for the potential protective role of vitamin K2 in Alzheimer’s disease.
Ask your neurologist if vitamin K2 is right for you because they keep proving it in science studies. All right. In 2021 study a study reached similar conclusions in a C6 cell line. Upon an increase in the concentration of vitamin K2 cells exhibited prolonged survival due to protection against amyloid beta induced neuronal death. Alzheimer’s induced death. This effect was reversible upon the addition of warfarin. They could cause those cell to die faster when they added warfarin which prevents vitamin K dependent carboxylation. So warfarin blocks vitamin K2 being used to prevent amyloid beta plaques and death of cells in your brain.
All right, we’re going to highlight something about warfarin here in just a second. Just hold on. I can’t believe they even decided to test warfarin here. Warfarin blocks vitamin K2. This blood thinner which can prevent dementia and Alzheimer’s. Vitamin K2 also reduce the number of oxidative stress in a dose dependent manner. The more vitamin K2, the less stress in the body. And at a concentration of 10 micromoles decreased by 2.5 fold the activity of caspase 3, an enzyme that mediates amyloid beta induced apoptosis. Caspase 3 causes more death of cells in the brain leading to Alzheimer’s.
Vitamin K2 decreased by two and a half times the activity of that enzyme that Causes worsening Alzheimer’s and dementia symptoms. Ask your doctor if vitamin K2 is right for you. Remember, they could block vitamin K2 in this cell line with warfarin. Anybody out there on a blood thinner called warfarin? Watch this. Let’s highlight warfarin. Stay with us, people. Warfarin blocks vitamin K2’s protective ability against Alzheimer’s and dementia. Read along with us, people. Warfarin drugs dot com. I have the website at the bottom. Warfarin FDA black box warning about warfarin. Warfarin can cause major or fatal bleeding.
Where did we see that before? Let’s focus on this. Warfarin can cause major or fatal bleeding. I’m going to crop that. Bring it up to the top of the screen. Warfarin can cause major or fatal bleeding and it blocks vitamin K2 that prevents Alzheimer’s and dementia. Do you remember this statement from earlier? Vascular dementia, which occurs because of microscopic bleeding in the brain, is the second most common cause of dementia in this presentation alone. This is now the third drug I’ve shown you which causes major fatal bleeding even in the brain. When you’re prescribed warfarin, ask them if fatal bleeding or worsening dementia from vascular dementia is right for you.
All right, that was just a side note. Let’s go back to the vitamin K2 study. These two studies both reached the conclusion that vitamin K2 suppresses the production of pro inflammatory cytokines, suggesting that vitamin K2 may have the potential to reduce brain inflammation and brain degeneration or death of cells in your brain neurodegeneration. By the way, Parkinson’s, Mississippi and Alzheimer’s are called neurodegenerative diseases. So if we go back, you’re gonna see vitamin K2 may have the potential to reduce neuroinflammation and block Parkinson’s, Ms. And Alzheimer’s diseases. Depression is both a risk factor for Alzheimer’s disease, depression being sad, and a comorbid condition that worsens the prognosis of Alzheimer’s disease.
Depression can precede dementia, depression can precede dementia and usually occurs in up to 50% of Alzheimer’s disease patients. I think it’s the opposite if you were losing your memory and recall and not remembering the faces of those in front of you. Have you ever seen the movie the Notebook? Oh, my God. It’s all about the beautiful side of Alzheimer’s. Oh, my God. What? All right. It is depressing to have Alzheimer’s or dementia symptoms. So yes. Why would you not be surprised if 50% of Alzheimer’s patients also have depression. One study in rats that had METS demonstrated that vitamin K2 prevents the development of anxiety and depression.
Oh, and just remember, if it happens in a mouse or rat, it’ll happen in a human. That’s what’s published by scientists. This paper has reviewed studies that consider the relationship between vitamin K2 and amyloid plaques, Alzheimer’s plaques, neurotoxicity, brain toxicity, brain inflammation and mitochondrial dysfunction, cognition, cardiovascular health, dysbiosis and Alzheimer’s disease death. When considered together, these studies strongly suggest that vitamin K2 could play an important role in Alzheimer’s disease prevention and therapy. Is your neurologist recommending this to your loved ones who have Alzheimer’s dementia? Are they showing you this information about vitamin K2 and vitamin D and if not, fire them.
And if you like them because you think they are sincere, you better tell them to sign up and be a Follower of the Dr. Ardis show or forward them this PowerPoint found in the resources tab of the Dr. Ardis Show. This PowerPoint you’re looking at right now is there for free to download and send or send them link to this podcast and they can watch and learn together with us all right in the Journal of Neuro Restoratology Restoring Brains and their health March 2024 just last year Magnesium Ions and Dementia Magnesium and Dementia Magnesium enhances learning and memory section 2.1 of the study Numerous studies show an association between magnesium and cognitive functions and that increasing magnesium level improves memory function.
Administration of 100 and 200 milligrams per kilogram of magnesium chloride enhanced memory for noxious stimuli in rats and an inhabit inhibitory avoidance training experiment. The administration of Magnesium sulfate at 270 milligrams per kilogram, a different form of magnesium during restation in rats ameliorated the impairment of learning ability induced by a poison in their offspring. Long term administration of water containing magnesium L Threonate was effective in increasing the magnesium content of the brain and it enhanced spatial learning, memory and working memory in rats. Remember, if it happens in a mouse or a rat, it’s going to happen in a human.
We have all these same genes. Magnesium levels in the brain enhanced fear memory in rats. Magnesium deficiency reduced fear memory in mice. Together, these studies suggest that magnesium plays a critical role in learning and memory functions. The magnesium deficiency can be caused by impaired absorption, abnormal distribution and increased exertion. Sweating it out, peeing it out, Pooping out Magnesium supplementation therapy significantly alleviates the switching phenomenon in Parkinson’s disease patients taking levodopa drugs. Dopamine drugs levodopa and taking magnesium supplementation ameliorates or resolves the difficult to control mental symptoms in Wilson’s disease. In addition, low magnesium can lead to metabolic impairment in dementia patients.
I repeat, not in rats or mice. Low magnesium can lead to a metabolic impairment in dementia patients. Parathyroid hormone secretion, which is behind your thyroid responsible for bone density primarily and immune health, is suppressed in dementia patients with low magnesium in section 3.3 of the same study Anti Inflammatory and Antioxidant Effects of Magnesium the inflammatory process plays a key role in Alzheimer’s disease disease processes Amyloid plaques are often surrounded by large numbers of activated astrocytes. Astrocytes are the same cells they call when they start exploding in number glioblastoma tumors and in microglial cells in the brain.
As the disease progresses, a large amount of amyloid plaque is deposited which leads to the overactivation of the surrounding microglia other cells in your brain, which releases a large number of inflammatory factors and cytokines that in turn exacerbate more amyloid plaque deposits, forming a vicious cycle that increases the speed of the progression of Alzheimer’s disease. Magnesium has a wide range of anti inflammatory effects and in lab studies they’ve been able to demonstrate that magnesium sulfate inhibits endothelial cell activation and blocks inflammatory responses through the NF Kappa beta pathways. You don’t need to know what NF Kappa beta pathways are.
Medical professionals and health professionals do. You don’t need to know that. Just know magnesium blocks it the inflammatory pathways. Magnesium protects cell membranes, it maintains mitochondrial function, it antagonizes excessive calcium influx and affects the synthesis and activation of antioxidant enzymes and non enzymatic antioxidants. Clinical that was a lot of scientific terminology. Just note it’s very beneficial for the body. Now magnesium in clinical and animal studies has shown that low magnesium induces oxidative stress and reduces antioxidant function in various tissues, producing excess oxygen radicals and superoxide anions and reduces the expression and activity of antioxidant enzymes. You want higher amounts of antioxidant enzyme activity to help prevent disease, Alzheimer’s included.
Oxidative stress is a major factor in tissue damage leading to dementia and therefore magnesium supplementation may inhibit the production of oxidative stress which is beneficial for Alzheimer’s and dementia, thereby reducing dementia mediated pathological damage and cognitive deficits. Nobody wants cognitive deficits, you want cognitive massive awesomeness. Now here’s another study on magnesium. The role of magnesium in depression, migraine, Alzheimer’s disease and cognitive Health A comprehensive review of multiple studies. The title in section 5 is the role of Magnesium in Dementia Prevention and Slowing the Progression of Alzheimer’s Disease. Let’s read together. Despite extensive research efforts, no effective cure currently exists for Alzheimer’s and dementia.
In this context, though, magnesium has garnered increased scientific interest due to its involvement in numerous neuro brain biological processes potentially influencing the onset and progression of Alzheimer’s disease. Although the precise mechanisms remain incompletely elucidated, we don’t know why. Recent studies have identified several pathways through which magnesium may exert a proliferative protective effect. Magnesium plays a critical role in maintaining synaptic plasticity and neuronal function. In order to have normal brain function, you have to have magnesium. It modulates the activity of N methyl D aspartate glutamate receptors, which are essential for learning and memory. You’re welcome. Did your neurologist tell your loved one who’s having dementia issues and Alzheimer’s that they need to be on magnesium and that it helps by improving the activity of N methyl D aspartate glutamate receptors which are essential for learning and memory? Did they tell you that? I doubt it.
Excessive activation of these receptors can lead to excit excitation Excitotoxicity. They excite the nerve so much they’ll die, a process that contributes to neuron damage and is implicated in Alzheimer’s disease progression. By attenuating or reducing leveling out NMDA receptor overactivation, magnesium may confer neuroprotective benefits. Brain Protective effects Magnesium exhibits antioxidant properties that may mitigate oxidative stress, an established contributor to Alzheimer’s disease. Oxidative stress arises from the excessive generation of reactive oxygen and nitrogen which damage the neuronal structures. Over time. Adequate magnesium levels may support endogenous antioxidant defenses and help counteract these harmful effects. The Regards study, a large long term American study, concluded that low blood levels of magnesium are associated with an increased risk of cognitive decline.
Magnesium concentrations in the upper range, in the upper normal range were associated with a protective effect. Ask your neurologist if magnesium is right for you. These findings suggest that maintaining adequate but not excessive magnesium levels may be linked to preserved cognitive health in older adults and younger. By the way, the atherosclerosclerosis risk in community studies also demonstrated that low midlife blood level of magnesium was associated with a 24% increased risk of developing dementia later in life compared to higher magnesium levels. Used as a reference, ask your doctor if you’re experiencing cognitive decline. If magnesium is right for you and then they stayed at the bottom Maintaining an adequate magnesium intake within a Recommended dietary allowance of 400-420mg per day for men, 310-320mg per day for women may play a particularly important role in the prevention of dementia, especially in older adults and individuals with metabolic risk factors such as diabetes or hypertension.
God, I hope all my followers are on magnesium. If you are not as excited as I get to see all of your nicotine badges on, I’d be more excited even to hear that you’re using magnesium every day and supplementing it every day. A growing body of scientific research supports the notion that magnesium is a critical modulator of neurodegeneration processes and may contribute to the prevention or slowing of Alzheimer’s disease progression. Again, ask your neurologist if magnesium is right for you. Now let’s dive into taurine, an amino acid all of you know I’ve educated people on glyphosate destroys in the ground plants in your body, which is very important for brain health.
Exploring Taurine’s potential in Alzheimer’s Treatment this is a comprehensive review. We’re just going to touch on a few highlighted statements here. In an experimental study, the researchers explored the therapeutic potential of taurine early stages of Alzheimer’s disease DEM dementia using an adult transgenic mouse model. Remember, if it happens in a mouse, it’s happening in a human. We all have the same genes. The mice were administered taurine orally at a dose of 1,000 milligrams for every 2.2 pounds of body weight for six weeks. The results showed that taurine supplementation led to improved behavioral performance in these Alzheimer’s disease mice.
Spatial memory was improved in these mice improved significantly after six weeks of taurine treatment, meaning they can move around in space. Even better, the passive avoidance test further supported the positive impact of taurine on behavior and hippocampal memory. These findings suggest that taurine may mitigate the damage caused by amyloid plaques and improve cognitive deficits. Amen. Drop the mic that’s called Alzheimer’s orally at a lower dose of 250 milligrams per kilogram a day. In this study, the results indicated improved spatial memory and enhanced MEM hypocypocampal memory in the taurine treated group, suggesting that lower doses of taurine may also have therapeutic benefits in Alzheimer’s disease.
Ask your neurologist if taurine is right for you and how many neurologists can you name or pinpoint or tag that is ever recommended. Taurine Please share this with all your neurologists. Taurine administration as pretreatment or treatment showed significant reductions in phosphorylated D tau protein levels with the highest dose of a hundred milligrams exhibiting the most pronounced effect when used prophylactically. This suggests that taurine may be more effective as a prophylactic measure against Alzheimer’s disease like mechanisms. Then another study revealed that taurine administration following a chemical called strepto streptozotosin injected in rats improved cognition and that STZ drug literally causes brain death.
Taurine at doses of 60mg and 120mg effectively mitigated cognitive impairment when they injected these chemicals inside of them and all of us are exposed to chemicals and heavy metals that are destroying our brain every day. These remember fluoride and it causing calcification of your pineal gland in your brain. That’s a chemical you might want to use. Taurine these findings suggest that chronic taurine treatment can attenuate or stop cognitive deficits in Alzheimer’s disease. Like models pre treatment with taurine resulted in improvement in cognitive function in one year old rats induced with this brain numbing chemical. Taurine exhibited a safeguarding effect against the detrimental effects of this toxic drug when administered prophylactically.
With all the chemtrails, with all the crap in our water, with all the crap in your food, you might want to consider taking taurine. They know these chemicals cause Alzheimer’s disease. Modeled animals, mammals and taurine when they gave it to them before they gave the actual chemical, it blocked it. Taurine has demonstrated neuroprotective properties in several ways. It has been shown to protect against mitochondrial dysfunction and nerve death caused by amyloid beta plaques. Additionally, taurine has been observed to interfere with amyloid aggregation or increased growth in numbers, further highlighting its potential therapeutic role in Alzheimer’s disease.
There was a study that suggested that taurine’s neuroprotective effects may be attributed to its ability to increase the blood flow production of hydrogen sulfide, which in turn decreases amyloid beta plaque levels and tau protein phosphorylation which leads to more amyloid plaques. Taurine has been recognized as an agonist of GABA receptors and experimental studies have shown that taurine can protect neurons from the toxic effects of amyloid beta plaques through GABA receptor activation. This GABAergic mechanism may play a crucial role in preventing and treating Alzheimer’s disease. Tell your neurologist what you learned here at the doctor the presence of lower taurine levels in the brain.
Lower levels of taurine in your cerebral spinal fluid and blood whoops. Of Alzheimer’s disease patients highlights a potential connection between taurine and and Alzheimer’s disease pathology, which is the progression of the disease. Studies affirm that taurine plays a crucial neuroprotective role by reducing endoplasmic reticulum stress, preventing oxidative stress, inducing apoptosis and guarding against the neurotoxic effects of amyloid beta. Conclusions. Through its multifaceted mechanisms, including mitochondrial protection, interference with amyloid aggregation and reduction of neuronal susceptibility to excitotoxic toxicity, which kills nerves, taurine emerges as a promising candidate for future therapeutic interventions. Aren’t you excited? You watched the Dr.
Ardis show as a potential player. As taurine advances as a potential player in Alzheimer’s disease therapeutics, it offers a glimmer of hope in improving the lives of those affected by this devastating condition. This is our new vitamin D3 plus K2. I just showed you the studies. Go back and watch the presentation. Download the PowerPoint how D3K2 has been proven. We now have it combined in organic form. Vitamin D3K2. Take one dropper full morning and night. Actually, just do it once a day if you want to. Remember, the goal was above 50 nanograms per millimoles of blood.
That’s a measurement you’ll see on any blood test. Here’s our magnesium complex supplement with 400 milligrams for every four capsules. Here’s our taurine, 500 milligrams in one capsule. Remember, you can scan the QR code to get notified here. Remember, you can also go to the resources tab at the doctor or show and download the PowerPoint here that you can share with your neurologist. Share with your loved ones who are dealing with dementia who are worried about losing their mind. This is our book moving beyond the COVID 19 lies. If you don’t own it, I’m sorry. You should have already.
You’re going to learn a lot from this book. All right, I want to get out of the presentation mode here. I had to push the button twice. All right, I’m Dr. Bryan Ardis. This is how not to lose your mind. When you’ve been diagnosed or been told that there’s a family history of concern of Alzheimer’s and dementia that awaits you if you’re already experiencing mild recall collapse or diminishment. If you’ve been diagnosed with mild cognitive impairment. I’m telling you, vitamin K2, vitamin D3, magnesium and taurine are proven over and over and over in medical research studies to improve the outcome in preserving your memory and cognitive functions.
It is my hope and prayer that the Dr. Dr. Ardis show can continue to bring you relevant information. Warnings. Oh, my goodness. Please tell everybody far and wide the drugs for Alzheimer’s and dementia are known to make the probability of more worsening cognitive decline and worsening dementia. I showed you why. Please tell all your friends how they’re going to lose their mind and the potential for that for everybody put on the drugs that they’re prescribing for Alzheimer’s and dementia. I’m Dr. Ardis Ardes. May this presentation bless your lives, is my prayer. We will see you next week on the Dr.
Ardis show. God bless.
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